NFAT1在脊髓背角小膠質(zhì)細胞中調(diào)節(jié)神經(jīng)病理性疼痛機制的研究（所屬項目其他成果）
1.G protein-coupled receptor GPR151 is involved in trigeminal neuropathic pain through the induction of G beta gamma/extracellular signal-regulated kinase-mediated neuroinflammation in the trigeminal ganglion（G蛋白偶聯(lián)受體GPR151通過誘導(dǎo)G β-γ/細胞外信號調(diào)節(jié)激酶介導(dǎo)的神經(jīng)炎癥而參與三叉神經(jīng)痛）
2.TLR8 and its endogenous ligand miR-21 contribute to neuropathic pain in murine DRG （TLR8（Toll樣受體）及其內(nèi)源性配體miR-21對小鼠DRG神經(jīng)性疼痛的作用）
3.Chemokine receptor CCR2 contributes to neuropathic pain and the associated depression via increasing NR2B-mediated currents in both D1 and D2 dopamine receptor-containing medium spiny neurons in the nucleus accumbens shell（趨化因子受體CCR2通過增加伏隔核殼中含有D1和D2多巴胺受體的中棘神經(jīng)元中NR2B介導(dǎo)的電流，促進神經(jīng)性疼痛和相關(guān)抑郁癥）
4.趨化因子CXCL10作為神經(jīng)病理性疼痛生物標(biāo)記物的研究
5.ZNF382 controls mouse neuropathic pain via silencer-based epigenetic inhibition of Cxcl13 in DRG neurons （ZNF382（鋅指蛋白轉(zhuǎn)錄因子）通過基于沉默者的表觀遺傳抑制DRG神經(jīng)元中的Cxcl13控制小鼠神經(jīng)性疼痛）
6.CXCL13通過CXCR5激活星形膠質(zhì)細胞中JNK參與痛覺過敏
7.Chemokines in chronic pain: cellular and molecular mechanisms and therapeutic potential（慢性疼痛中的趨化因子：細胞和分子機制以及治療潛力）
8.MMP24 Contributes to Neuropathic Pain in an FTO-Dependent Manner in the Spinal Cord Neurons （基質(zhì)金屬蛋白酶24 在脊髓神經(jīng)元中依賴于FTO（肥胖調(diào)節(jié)基因，一種RAN 6-甲基腺苷（m6A）脫甲基酶，促進腫瘤細胞的糖酵解，抑制T細胞活性，影響功能，幫助腫瘤實現(xiàn)免疫逃避）方式促進神經(jīng)性疼痛）
9.慢性疼痛的分子機制和鎮(zhèn)痛新靶點研究
10.Increased CXCL13 and CXCR5 in Anterior Cingulate Cortex Contributes to Neuropathic Pain-Related Conditioned Place Aversion （前扣帶回中CXCL13和CXCR5的增加有助于神經(jīng)性疼痛相關(guān)的條件性位置規(guī)避）
11.Demethylation of G-Protein-Coupled Receptor 151 Promoter Facilitates the Binding of Kruppel-Like Factor 5 and Enhances Neuropathic Pain after Nerve Injury in Mice （G蛋白偶聯(lián)受體151啟動子的去甲基化促進Kruppel樣因子5的結(jié)合并增強小鼠神經(jīng)損傷后的神經(jīng)性疼痛）
12.Intravenous Administration of Triptonide Attenuates CFA-Induced Pain Hypersensitivity by Inhibiting DRG AKT Signaling Pathway in Mice （靜脈注射雷公藤內(nèi)酯醇通過抑制DRG AKT信號通路減輕CFA誘導(dǎo)的小鼠疼痛超敏）
13.Spinal CXCL9 and CXCL11 are not involved in neuropathic pain despite an upregulation in the spinal cord following spinal nerve injury （盡管在脊髓神經(jīng)損傷后，脊髓中的 CXCL9和 CXCL11表達上調(diào)，但它們并不參與神經(jīng)性疼痛）
