IL-8 (CXCL8)

白細胞介素?8（IL-8，IL8，也稱為?CXCL8）是一種由不同類型的細胞產(chǎn)生的促炎細胞因子，包括單核細胞、巨噬細胞和內(nèi)皮細胞。炎癥刺激，如?LPS，強烈誘導單核細胞產(chǎn)生?IL-8。

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艾美捷抗人IL-8 (CXCL8)單抗MT8F19：

這種單克隆抗體能夠在?ELISA?中靈敏和特異地檢測人?IL-8。

艾美捷推薦?MT8F19?作為?ELISA?中的檢測抗體與捕獲抗體?MT8H6?結(jié)合使用。

艾美捷抗人IL-8 (CXCL8)單抗MT8F19配套研究：

3310-8-1000 ELISA?偶聯(lián)物：鏈霉親和素-ALP 1 ml

3310-9-1000 ELISA/ELISpot?偶聯(lián)物：Streptavidin-HRP 1 ml

3560-1-250?抗人?IL-8 (CXCL8) mAb (MT8H6/8F19)，未偶聯(lián)?250 μg

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艾美捷抗人IL-8 (CXCL8)單抗MT8F19參考文獻：

Arruda-Silva, F., et al. (2017). Human Neutrophils Produce CCL23 in Response to Various TLR-Agonists and TNFalpha. Frontiers in cellular and infection microbiology.

Castellucci, M., et al. (2015). IL-10 disrupts the Brd4-docking sites to inhibit LPS-induced CXCL8 and TNF-alpha expression in monocytes: Implications for chronic obstructive pulmonary disease. The Journal of allergy and clinical immunology.

Panda, S. K., et al. (2018). Galectin-9 inhibits TLR7-mediated autoimmunity in murine lupus models. The Journal of clinical investigation.

Skrlec, K., et al. (2017). Evasin-displaying lactic acid bacteria bind different chemokines and neutralize CXCL8 production in Caco-2 cells. Microb Biotechnol.

Tamassia, N., et al. (2018). A Reappraisal on the Potential Ability of Human Neutrophils to Express and Produce IL-17 Family Members In Vitro: Failure to Reproducibly Detect It. Front Immunol.